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International Journal of Advance Research in Medical Surgical Nursing

P-ISSN: 2663-225X, E-ISSN: 2663-2268, Impact Factor: RJIF 5.3
International Journal of Advance Research in Medical Surgical Nursing
Printed Journal   |   Refereed Journal   |   Peer Reviewed Journal

2024, Vol. 6, Issue 1, Part A

Anti-neurofascin demyelinating diseases (Anti-NF diseases)

Nandeesh Kumar PR

Neurofascins are proteins crucial in nerve function; when the body's immune system attacks these proteins, disorders known as anti-neurofascin demyelinating diseases occur. Both the central and peripheral nervous systems are susceptible to injury from these assaults to the myelin sheath that surrounds nerve fibers. Prominent medical problems associated with anti-NF diseases include Multiple Sclerosis (MS), Neuromyelitis Optica (NMO), and Chronic Inflammatory Demyelinating Polyneuropathy (CIDP). The combination of clinical indicators, blood testing, and imaging studies is used to diagnose anti-NF disorders. The intensity of the symptoms and the particular disease determine the course of treatment. Medications that alter the immune system, medications that halt the progression of the illness, occupational and physical therapy, pain management, and supportive care are a few of the treatments. In the treatment of patients with anti-NF illnesses, nurses are essential. They offer patients all-encompassing treatment that attends to their requirements on a social, emotional, physical, and educational level. Nursing responsibilities include patient safety, communication and advocacy, long-term planning, symptom management, immunemodulatory therapies, rehabilitation and adaptive techniques, evaluation and monitoring, education and support, and documentation.
Pages : 26-29 | 205 Views | 95 Downloads


International Journal of Advance Research in Medical Surgical Nursing
How to cite this article:
Nandeesh Kumar PR. Anti-neurofascin demyelinating diseases (Anti-NF diseases). Int J Adv Res Med Surg Nurs 2024;6(1):26-29. DOI: 10.33545/surgicalnursing.2024.v6.i1a.164
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